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OCD, anxiety, PANDAS and PANS: Dr Brandon Brock

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This is a quick reminder that The Autism, ADHD and Sensory Processing Disorder Summit starts tomorrow.

I really enjoyed Dr. Brandon Brock’s interview, Understanding PANS and PANDAS role in ASD, ADHD and SPD, and it is particularly relevant for anxiety and OCD. During the interview he describes what he often finds with these children with PANS (Pediatric Acute-onset Neuropsychiatric Syndrome) and PANDAS (Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcal Infections):

A lot of times the child may come in having nightmares or they just start bed wetting or they’re having fears or a little bit of irritability and they get blamed on all kinds of things. You know they get blamed on maybe a sketchy home environment that’s really not that sketchy, or maybe they just say it’s normal for a child to go through this, or maybe they’re just starting to hit puberty so they’re getting more aggressive. In other words there’s always a reason other than looking at the brain physiology. Why is it changing? Is the brain physiology changing because maybe there is an environmental factor? You know maybe there is some abuse or something. And that does happen.

But on a bigger scale maybe they had some sort of infectious disease, or maybe they had a toxin that got into their body, and it made their immune system react, and it started giving them symptoms like abnormal movements, ticks, especially in the face, lip smacking, hair twirling.

He talks about how these immune reactions to the brain and basal ganglia cause symptoms:

So when you have an immune response against the basal ganglia it’s kind of like pushing the play button over and over and over again. So now we see things like obsessive compulsive disorder, we see tics, we see choreiform movements [repetitive and rapid, jerky, involuntary movement that appears to be well-coordinated], we see nightmares and bizarre thoughts, or the kid does something that we call a perseveration. It is the repetition of a particular response (such as a word, phrase, or gesture), so they say something over and over and over. So there’s a fine line between a kid being a kid and then a kid having something like PANDAS. And really what we call that is auto-immune encephalitis, which just means inflammation of the brain. So the kid’s brain really is kind of like, if you want to say it metaphorically, on fire.

Dr. Brock talks about the 2 strep tests or anti-streptococcal antibody titers that are commercially available and determines whether the child has had a previous strep infection:

  • Antistrepolysin O (ASO) titer,* which rises 3-6 weeks after a strep infection, and
  • Antistreptococcal DNAse B (AntiDNAse-B) titer, which rises 6-8 weeks after a strep infection.]

In addition to the many other labs tests they do, he goes on to discuss another whole set of antibodies that can become positive down the road:

Dopamine antibodies, calcium calmodulin mechanisms, and then all of the intra-cellular structures, like the tubulin structures, the alpha and beta tubulin structures, the micro-tubulin structures, and even the cell wall. So we have an antibody panel that really says it’s either the structure, it’s either the receptor, or it’s the actual pumping mechanism that makes dopamine.

He didn’t mention this but it’s the Cunningham Panel done by Moleculera Labs

Dr. Brock goes on to talk about functional neurology, diet, how to find a good practitioner and much more. It’s a great interview!

The Autism, ADHD and Sensory Processing Disorder Summit, hosted by Tara Hunkin, NTP, runs from June 19-28, 2017.

It will be 10 days of eye opening information into the root causes of your child’s neurological dysfunction. Imagine learning about what may have caused their symptoms and how to address them with nutrition and biomedical approaches and leverage the power of positive neuroplasticity to improve function, health and their lives.

As I mentioned in the prior summit announcement many of the interviews on this summit may be applicable for you even if you don’t have a child with a sensory processing disorder, ADHD and/or autism – many of my clients with anxiety often benefit from the biomedical support that many of these speakers are addressing. Simply replace sensory processing disorder, ADHD and/or autism with anxiety and listen and learn.

And if you’re new to my work, do also tune into my interview: Anxiety’s Role in ASD, ADHD and SPD and how nutrient therapy can help.

Here are a few other speakers and their interesting topics (and I can’t wait to hear them all):

  • David Perlmutter, MD: The role of the microbiome in neurological health.
  • Alex Doman: Using music to heal your child’s brain
  • Derrick MacFabe, MD: The role of propionic acid in the multi-system challenges found in ASD.
  • Sonia McGowin, DC: How to know if biomedical intervention is right for your child.

You can register for The Autism, ADHD and Sensory Processing Disorder Summit here

Anxiety in autism, ADHD and sensory processing disorders

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I had the pleasure of being interviewed by Tara Hunkin on the upcoming Autism, ADHD and Sensory Processing Disorder Summit and share my expertise on anxiety since we so often see anxiety in this population. My topic is: Anxiety’s Role in ASD, ADHD and SPD and how nutrient therapy can help.

I start out sharing some of the research on the incidence and connections.

This 2009 paper Sensory Overresponsivity and Anxiety in Children With ADHD in the American Journal of Occupational Therapy, reports that:

Approximately 25% of children with attention deficit hyperactivity disorder (ADHD) have a comorbid anxiety disorder.

The purpose of this particular study was to determine whether sensory overresponsivity (SOR) or sensory processing disorders are related to increased anxiety in children with ADHD. There were twenty-four children between the age of 6 to 10 with ADHD and 24 children without ADHD.

The study concluded that:

Children in the ADHD + SOR [sensory over responsivity] group were significantly more anxious than both the ADHD-only and non-ADHD (control) groups.

Occupational therapists treating children with ADHD and SOR should be aware that these children may also have anxiety and discuss options with families for prevention or treatment.

I would add that anyone working with these children should be aware of the connections: doctors, nutritionists, psychologists and anyone else on the health team.

We also discuss this 2012 paper, also from the American Journal of Occupational Therapy – Sensory Overresponsivity and Anxiety in Typically Developing Children and Children With Autism and Attention Deficit Hyperactivity Disorder: Cause or Coexistence?

Reviews of the coexistence of ASD [autism spectrum disorder] and anxiety disorders have concluded that among children and youth with ASD, anxiety disorders are highly prevalent, clinically significant, and varied as to specific type of anxiety disorder

Approximately 25% of children with attention deficit hyperactivity disorder (ADHD) also have anxiety disorder, a rate that is elevated when ADHD is seen in conjunction with conduct or oppositional defiant disorders

The paper mentions these 3 factors as models worthy of further study in order to understand the relationship between anxiety, sensory overresponsivity, autism and ADHD:

(1) anxiety caused by, or a symptom of, SOR (primary anxiety model);
(2) SOR caused by, or a symptom of, anxiety (primary SOR model); and
(3) the presence of both anxiety and SOR, linked by way of another factor

Based on what I know about anxiety I feel it could be a combination of all of the above.

My interview also covers the following around how to address the anxiety in these children and their moms (who also often have anxiety):

  • Low serotonin anxiety, symptoms, using tryptophan and 5-HTP and precautions with using them with an SSRI
  • A young girl with RAD (reactive attachment disorder) with rage/anger, anxiety, insomnia, and sugar cravings and the successful use of chewable tryptophan, addressing low iron and a gluten-free diet
  • A young boy with OCD and the successful use of both tryptophan and inositol
  • Low GABA anxiety, the use of GABA and not phenibut, and cautions about using too much
  • A young girl with ADHD and irritability and the successful use of GABA
  • Pyroluria incidence and symptoms and how it ties back to neurotransmitter imbalances

Sensory processing disorders, ADHD and autism are not my expertise and I don’t work much with children so I’m really pleased to be bringing you this information via the other amazing speakers AND listening and learning myself!

Here are some speakers and topics I’m particularly interested in

  • Brandon Brock, RN, DC: Understanding PANS and PANDAS role in ASD, ADHD and SPD.
  • Elizabeth Mumper, MD: Mitochondrial Dysfunction: What it is and how to address the underlying causes.
  • Richard Frye, MD, Ph.D.: Cerebral Folate Deficiency: and how it impairs neurological health.
  • Nancy O’Hara, MD: What you need to know about cell danger response in ASD & other neurodevelopmental disorders.
  • John Tjenos, NTP: The importance of the vagus nerve and how to build vagal tone with essential oils.

We have so much to learn from practitioners and researchers working in this area. And children affected by these conditions do recover!

The Autism, ADHD and Sensory Processing Disorder Summit, hosted by Tara Hunkin, NTP, runs from June 19-28, 2017.

It will be 10 days of eye opening information into the root causes of your child’s neurological dysfunction. Imagine learning about what may have caused their symptoms and how to address them with nutrition and biomedical approaches and leverage the power of positive neuroplasticity to improve function, health and their lives.

Do also keep in mind that these topics may have relevance for you even if you don’t have a child with a sensory processing disorder, ADHD and/or autism. These children are the canaries in the coal mine and many of my clients with anxiety can benefit from the biomedical support that many of these speakers are addressing. Simply replace sensory processing disorder, ADHD and/or autism with anxiety and listen and learn.

You can register for The Autism, ADHD and Sensory Processing Disorder Summit here

Feel free to ask questions or provide feedback and your experiences in the comment section below.

GABA for children: ADHD, focus issues, irritability, anxiety and tantrums

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Is GABA safe for children with symptoms of ADHD? I was asked this question after my interview on anxiety on the Diabetes Summit. I shared the question and my answer on facebook:

I’ve used GABA (and theanine) very successfully with children with physical anxiety symptoms and ADHD. I’ve also used tyrosine for the attention/focus issues. Plus I always consider gluten, blood sugar control (animal protein at breakfast often makes the biggest difference), colors/additives, low iron, low zinc and candida (this list is not inclusive but is a good starting point)

Jenny is a mom in my facebook community and responded to the above post, sharing that a friend of hers recommended GABA for her daughter after reading some of my posts. She shared this wonderful feedback:

My daughter hasn’t been diagnosed with ADHD but has a lot of ADHD qualities. We were having a huge amount of behavior problems as she is getting older (she’s 11). I did some research and went to the health food store and bought a bottle. At this point I was mentally exhausted from all the fighting and drama at home and at school. I was desperate and didn’t want to put her on any hard medication.

She has had amazing behavior at school and at home since giving it to her. She’s almost like different child. GABA has truly changed our life. She’s been taking it for almost 2 months.

What wonderful results! I’m thrilled for Jenny and her daughter. I asked Jenny to share what her daughter’s symptoms/behaviors were before they used the GABA and which symptoms improved. This is what Jenny shared:

…before the GABA she was irritable, she was fighting with her teacher and schoolmates, she couldn’t focus and was distracted by anything and everything, she was constantly disrupting the class and she was throwing major tantrums over ridiculous stuff (like her hair didn’t do what she wanted), she was mouthy and everything was a fight.

We are not seeing 99% those behaviors at all anymore. She of course is still your typical pre-teen but if I say “no you can’t have that” our “no you can’t go there” her reaction is OK. Before it would have been a major fight or meltdown. Her teacher is reporting to me every day about her wonderful days.

I love hearing this feedback and it’s not unusual for children to act out when they have low GABA levels. Adults may identify with being anxious, but children may appear irritable, unfocused and distracted, may have tantrums and meltdowns, and all the while not be able to articulate how they are feeling.

Long–term use of GABA and root causes of low GABA

Jenny also posed some additional questions about GABA:

I do have some questions – I can’t really find anything online saying that it’s OK to give to kids. I mean, I’ve seen the results but it still worries me. She takes one 25 mg pill a day – do you think that’s OK?? Should I give her more or less or keep it the same? Just wondering your opinion?

I shared some of this feedback on the facebook thread and have elaborated on it here on the blog.

There is no research on children and the long-term use of amino acids but plenty of clinical evidence showing they can be used safely for 3-12 months. But I am also always working with my clients to find out why there is the deficiency in the first place and addressing that. This could include any or all of the following:

  • low zinc/vitamin B6/low iron which are all co-factors needed to make GABA (and then I’d want to know why are these low)
  • dysbiosis, parasites and/or candida (we make GABA in the gut and we may have GABA-eating bacteria in the gut – such as gabavorous)
  • too much stress at school, playing sports and/or at home
  • not getting enough sleep
  • not eating animal protein at breakfast (this affects blood sugar control and can stress the adrenals and impact cortisol, sex hormones and brain chemicals)
  • gluten sensitivity (gluteomorphins in gluten can impact mood and gluten can damage the gut leading to nutritional deficiencies)
  • low animal protein intake and/or eating a low-fat diet
  • low stomach acid (so you can’t digest the protein you’re eating)
  • toxin exposure (like pesticides or BPA)
  • low total cholesterol (caused in part by low levels of enzymes for fat digestion or a low-fat diet)

I shared these 60+ Nutritional and Biochemical Causes of Anxiety on my closing call of the The Anxiety Summit season 3.   I use this to help put the puzzle pieces together for my clients to resolve their anxiety and related issues like ADHD, insomnia, sadness, irritability etc.

When Jenny saw some of the root causes for low GABA she shared that she’s going to be making an appointment with her doctor. Keep in mind that in this instance it will need to be a functional medicine doctor or nutritionally trained doctor or a doctor who is open to learning about all this. I always also suggest taking a copy of my book The Antianxiety Food Solution to share with the doctor too!

What GABA product?

I typically have clients start with Source Naturals GABA Calm which contains 125mg of GABA. I was curious to know which 25mg GABA product she was using. It turns out to be a sublingual product by KAL.

  

 

You can find the other GABA products I use and recommend here and the precautions with using GABA here. Typically it’s an amino acid that the least issues, unless too high an amount is used.   I often hear that 500mg or 1000mg was used and caused dizziness or breathlessness – these are clues it’s too much!

I asked Jenny if I could share here to give other mom’s hope and inspiration and practical results so they can help their children – and really I appreciate her being willing to do so.

Thanks for sharing Jenny! Happy mama and happy daughter – it warms my heart to hear stories like this!

Have you used GABA and observed a reduction or elimination of ADHD, irritability or anxiety?

If you’re a practitioner, have you seen results like this with your clients or patients?

The Anxiety Summit – What if… Nutrition could Treat Anxiety and Depression?

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Julia Rucklidge PhD, Professor of Clinical Psychology in the Department of Psychology at the University of Canterbury, Christchurch, New Zealand, was interviewed on the Anxiety Summit by host of the Anxiety Summit, Trudy Scott, Food Mood Expert and Nutritionist, author of The Antianxiety Food Solution.

Julia has over 75 peer-reviewed studies and is passionate about helping people find alternative treatments to medications for their psychiatric symptoms. Her current research interests can be found at the University of Canterbury site here.

What if… Nutrition could Treat Anxiety and Depression?

  • The increasing prevalence of mental health problems and medication side-effects
  • Why diet is so important when it comes to mental health (anxiety, depression, ADHD and even schizophrenia)
  • Nutritional medicine as mainstream in psychiatry and ISNPR (International Society for Nutritional Psychiatry Research)
  • The problems with food: pesticides, soil quality, Roundup
  • Micronutrient research for ADHD, anxiety, bipolar and PTSD
  • The emerging field of nutritional mental health: Inflammation, the microbiome, oxidative stress, and mitochondrial function

Here are some snippets from our interview:

Why is nutrition important? As a scientist I can just say because the data says it’s important. So if we start with the wonderful work that has really been spearheaded from Felice Jacka and there are other people in Spain and in Japan who have published similar studies showing that the more you eat a processed, western type of diet, the higher your risk for mental illness. They’ve published data on depression and anxiety, but also other mental illness as well. And the more you eat what we call, a prudent or a healthier Mediterranean type of diet, the lower your risk for depression and anxiety.

How long is it going to take our society to pay attention to the research that shows that suboptimal nutrition is contributing to the epidemic of mental illness? Are we just going to sit around and ignore this evidence to our peril? Or are we going to start paying attention and start to invest in the really important research that needs to happen?

Here is an earlier blog post I did on Dr. Rucklidge’s excellent TEDX talk: The surprisingly dramatic role of nutrition in mental health

Here is part of the recent paper titled “What if nutrients could treat mental illness?”

We are at a tipping point in psychiatry.

The growing body of literature on the effect of nutrients on mental health is compelling enough and consistent enough for us to pay attention. It is time to revisit the role of diet and supplementary nutrients in the treatment of mental illness and to invest in this line of research.

Here is the smoking and cannabis study we talked about: Use of micronutrients attenuates cannabis and nicotine abuse as evidenced from a reversal design: a case study.

This case adds to a growing body of research supporting the use of micronutrients in the treatment of psychiatric symptoms and suggests it may extend to substance dependence. Micronutrients, by assisting with mood regulation and reductions in anxiety, may assist with successful cessation of drug use.

We discussed this earthquake paper, a favorite of hers: Shaken but unstirred? Effects of micronutrients on stress and trauma after an earthquake: RCT evidence comparing formulas and doses.

This study supports micronutrients as an inexpensive and practical treatment for acute stress following a natural disaster with a slight advantage to higher doses

If you are not already registered for the Anxiety Summit you can get live access to the speakers of the day here: www.theAnxietySummit.com

Missed this interview or can’t listen live? Or want this and the other great interviews for your learning library? Purchase the MP3s or MP3s + transcripts and listen when it suits you.

You can find your purchasing options here: Anxiety Summit Season 1, Anxiety Summit Season 2, and Anxiety Summit Season 3.

Pyroluria and chronic fatigue syndrome: is there a link?

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tired-boyLast week I blogged about the social anxiety condition called pyroluria (Pyroluria, high mauve, pyrrole disorder, malvaria, elevated kryptopyrroles and social anxiety) and received some great comments on this and the other pyroluria blogs.

One question on this blog: The Anxiety Summit: How zinc and vitamin B6 prevent pyroluria and social anxiety was related to Chronic Fatigue Syndrome/CFS so I’ve decided to share this and some additional information I was able to find.

Hi Trudy, I’m calling from Melbourne Australia, I was wondering if you have had any of your clients present with Chronic Fatigue Syndrome as well as pyroluria. I have just recently been diagnosed with CFS, I also have ADHD. Someone on a CFS forum that I belong to told me about pyroluria and said it is common in ADHD and CFS sufferers. While the link between ADHD and pyroluria is well documented, I haven’t been able to find any information regarding a connection between Chronic Fatigue Syndrome and pyroluria. I notice that there is an overlap of symptoms in the two conditions. I have all of the symptoms of CFS and many of the symptoms of pyroluria. I’m very interested in hearing your views about CFS and how it relates to pyroluria if at all. — Tom

I was not aware of a CFS/pyroluria connection until now but see there is some research showing how CFS and mood disorders/anxiety can have related causes and one of them is inflammation:

An intriguing and hitherto unexplained co-occurrence: Depression and chronic fatigue syndrome are manifestations of shared inflammatory, oxidative and nitrosative (IO&NS) pathways

Low zinc is a factor in pyroluria and depression and of course we may also see low zinc in CFS:

Lower serum zinc in Chronic Fatigue Syndrome (CFS): relationships to immune dysfunctions and relevance for the oxidative stress status in CFS

I find this paper very interesting since many of these same nutrients are commonly low in folks with anxiety and depression: Nutritional strategies for treating chronic fatigue syndrome

A detailed review of the literature suggests a number of marginal nutritional deficiencies may have etiologic relevance. These include deficiencies of various B vitamins, vitamin C, magnesium, sodium, zinc, L-tryptophan, L-carnitine, coenzyme Q10, and essential fatty acids.

I’m surprised the above paper didn’t mention iron anemia.  This is very common with pyroluria.  This paper: Iron insufficiency and hypovitaminosis D in adolescents with chronic fatigue and orthostatic intolerance found this

In patients presenting with chronic fatigue and/or orthostatic intolerance, low ferritin levels and hypovitaminosis D are common

These are just a few of the links I found by doing a very quick pubmed search. There are likely many more.

Everything is so connected and inter-related! And it’s interesting how certain deficiencies can manifest in certain ways – one person may find themselves with a CFS diagnosis, someone else with arthritis and yet someone else with heart disease. I think we need to be thinking about addressing nutrient deficiencies, balancing biochemistry and getting healthy, perhaps more just than addressing a diagnosis.  

If you score high on the Pyroluria Questionnaire I would suggest simply addressing the pyroluria which may have some ripple down effects and actually improve the symptoms of CFS.

Now the next post will have to dig into the link between ADHD and pyroluria. Stay tuned.

In the meantime, I’d love to see your comments/questions if you can relate to any of this. Also, please do share in the comments if you know you have pyroluria and find the nutrients have eliminated your social anxiety and inner tension symptoms, and have also helped your CFS.

 

Organic foods, Stanford study, pesticides and the brain

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Conventional peppers are highly sprayed with pesticides

We should be concerned about non-organic produce and pesticides despite the recent research published by Stanford: Smith-Spangler C, Brandeau ML et al. 2012. Are Organic Foods Safer or Healthier Than Conventional Alternatives?: A Systematic Review. Annals of Internal Medicine. 2012;157(5):348-366

There are two parts to this study:

  • the study states there is no difference between the nutrient values in organic vs non-organic produce : “The published literature lacks strong evidence that organic foods are significantly more nutritious than conventional foods.” However it appears some studies that do show differences were not included in the review – see Jeff Cox’s blog below
  • the study recognizes that pesticides are present in non-organic produce but that they are possibly nothing to be concerned about. I disagree with this and will show you the evidence.

The focus of this article is to address this statement in the conclusion: “Consumption of organic foods may reduce exposure to pesticide residues…” and why it’s important.

Let’s look at the some of the research around pesticide exposure and how this can affect the brain, cognition, ADHD, anxiety and depression in particular.

Mearns J, Dunn J et al. 1994. Psychological effects of organophosphate pesticides: A review and call for research by psychologists. Journal of Clinical Psychology 50(2):286-294.

Organophosphates (a commonly used toxic pesticide) “act directly on the nervous system by inhibiting the neurotransmitter acetylcholine.” They have “acute psychological and behavioral effects, such as anxiety, depression, and cognitive impairments.” The researchers suggest that long-term psychological effects of low-level exposure have not been determined satisfactorily.

Mackenzie Ross SJ, Brewin CR et al. 2010. Neuropsychological and psychiatric functioning in sheep farmers exposed to low levels of organophosphate pesticides. Neurotoxicoly and Teratology.32(4):452-459.

In this study sheep framers exposed to low levels of organophosphate pesticides experienced increased anxiety and depression, as well as worse cognitive function and memory.

Bouchard MF, Bellinger DC et al. 2010. Attention-deficit/hyperactivity disorder and urinary metabolites of organophosphate pesticides. Pediatrics 125(6):e1270-1277.

More recently, this study found that even low levels of pesticides in conventionally grown vegetables and fruit increased the risk of developing attention deficit/hyperactivity disorder in 8 to 15 years of age.

Cherry N, Burstyn I et al. 2012. Mental health in Alberta grain farmers using pesticides over many years. Occup Med (Lond) 62(6):400-6.

When it comes to elderly grain farmers in Alberta those “with mental ill-health in hospital discharge records were more likely to have been exposed to phenoxy compounds for ≥35 years.”

Coleman MD, O’Neil JD et al. 2012. A preliminary investigation into the impact of a pesticide combination on human neuronal and glial cell lines in vitro. PLoS One. 7(8):e42768.

This study found that certain fungicides (pyrimethanil, cyprodinil and fludioxonil), alone and also in combination “showed significant reductions in cellular ATP.” “The effects on energy metabolism were reflected in their marked toxic effects on mitochondrial membrane potential.” There was also “evidence of oxidative stress.” The authors conclude: “This report suggests that the impact of some pesticides, both individually and in combinations, merits further study in terms of their impact on human cellular health.”

Braquenier JB, Quertemont E et al. 2010. Anxiety in adult female mice following perinatal exposure to chlorpyrifos. Neurotoxicol Teratol. 32(2):234-9.

In this study “chronic perinatal exposure to low doses” of insecticides led to an increase in anxiety in the offspring of female mice. The authors suggest that “the routes of administration and the duration of exposure during brain development may be factors to consider when studying the development of anxiety.”

Malhotra A, Nair P et al. 2011. Efficacy of zinc as a nutritional supplement in ameliorating chlorpyrifos-induced neurotoxicity in rats. J Environ Pathol Toxicol Oncol. 2011;30(3):225-33.

This animal study found that pesticide exposure resulted in an “increase in the levels of lipid peroxidation and reactive oxygen species in both cerebellum and cerebrum.” Also, glutathione (a potent detoxifier) was decreased and anxiety levels were increased. The interesting thing is that researchers concluded that zinc (one of my favorite nutrients for anxiety and depression) “has potential to act as a neuroprotectant against pesticide-induced neurodegenerative and behavioral disorders but further investigations need to be conducted to understand the exact mechanism of neuroprotection.”

I’m encouraged by this research and look forward to further studies that will examine the impacts of pesticides on the nervous system. So yes, we obviously do need to be concerned about pesticides in the produce we consume. This August 2012 Neurotoxicology paper states it well: “The association between pesticide exposure and neurobehavioral and neurodevelopmental effects is an area of increasing concern .”

Footnote and additional reading:

This is the headline of one of the original Stanford blogs: Little evidence of health benefits from organic foods, Stanford study finds.

Much of the media used this and ran with it. Here is the article on ScienceDaily.com  NPR’s food blog, The Salt took the same approach as did Huffington Post and others.

Thank you France Moore Lappe for your wisdom: Stanford Scientists Shockingly Reckless on Health Risk And Organics.   And Mike Adams, the Health Ranger writes: Flawed organic foods study really just a media psyop to confuse the public about organics while pushing GMOs.   Jeff Cox, shares some of the nutrient research that seems to be missing from the Stanford study in his blog:  Monsanto’s Tricky Plan to Defeat GMO Labeling?